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Seminars and Events > The actin-bundling protein plastin increases cortical connectivity to promote robust polarization and timely cytokinesis in early C. elegans embryogenesi
Past Events
Category |
Seminar |
Date and Time |
2017-09-08 14:00 - 15:00 |
Venue |
Seminar Room A7F |
Speaker |
Wei Yung Ding |
Affiliation |
Mechanobiology Institute, National University of Singapore |
Title |
The actin-bundling protein plastin increases cortical connectivity to promote robust polarization and timely cytokinesis in early C. elegans embryogenesi |
Poster |
click here to download(PDF) |
Host |
Li Kun Phng |
Abstract |
Maintenance of the animal cell shape by the actomyosin cortex underlying the plasma membrane and generation of the contractile force within the cortex by non-muscle myosin II drive cellular morphogenetic processes such as gastrulation and cell division. Nevertheless, the regulation of the architecture and dynamics of the cortex by actin-bundling proteins is still poorly understood. Here we demonstrate that the evolutionarily conserved actin-bundling protein plastin (a.k.a fimbrin) is instrumental for the generation of potent cortical actomyosin contractility in the C. elegans zygote. Endogenous plastin, labelled by GFP via CRISPR/Cas9 knock-in, was found to be associating with both formin- and arp2/3-mediated subpopulations of F-actin, and enriched in contractile structures. Plastin was required for effective coalescence of nascent NMY-2 filaments into large contractile foci and for long-range coordinated contractility in the cortex. In the absence of plastin, force-dependent cellular processes during early embryogenesis, i.e. polarization and cytokinesis, were both significantly compromised, and 50% of embryos died during development. Based on these results as well as in silico CytoSim simulations, we propose that by increasing the connectivity of the F-actin meshwork, plastin enables the cortex to generate stronger and more coordinated forces to execute cellular morphogenesis. |
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